Harvard University Medical School Researchers find that Energy Healing is Cytotoxic to Pancreatic Cancer Cells But Not to Normal Cells

The online medical dictionary defines the term 'cytotoxic' as follows:

"Cytotoxic: Toxic to cells, cell-toxic, cell-killing. Any agent or process that kills cells. Chemotherapy and radiotherapy are forms of cytotoxic therapy. They kill cells."

In the June 2006 edition of the International Journal of Biochemistry & Cell Biology in an article entitled "External Qi of Yan Xin Qigong differentially regulates the Akt and extracellular signal-regulated kinase pathways and is cytotoxic to cancer cells but not to normal cells" several Harvard University Medical School researchers found energy healing ('external qi' healing in qigong lexicon) to be cytotoxic to pancreatic cancer cells but not to normal cells.

The authors had the following to say about traditional approaches to pancreatic cancer care:

"Carcinoma of the pancreas is the fifth leading cause of cancer-related deaths in Western countries, with an overall 1-year survival rate of ~12% and 5-year survival rate of 3-5%. Resistance to chemotherapy is a major cause of treatment failure and poor prognosis in pancreatic cancer."

The study states that "long-term clinical observations and ongoing studies have shown significant antitumor effect of external Qi of Yan Xin Qigong which originated from traditional Chinese medicine."

Furthermore "a single 5 min exposure of BxPC3 cells to external Qi of Yan Xin Qigong induced apoptosis, accompanied by a dramatic increase of the sub-G1 cell population, DNA fragmentation, and cleavage of caspases 3, 8 and 9, and poly(ADP-ribose) polymerase. Prolonged treatment with external Qi of Yan Xin Qigong caused rapid lysis of BxPC3 cells. In contrast, treatment of fibroblasts with external Qi of Yan Xin Qigong induced transient activation of extracellular signal-regulated kinases and Akt, and caused no cytotoxic effect. [LAYPERSON TRANSLATION:] These findings suggest that external Qi of Yan Xin Qigong may differentially regulate these survival pathways in cancer versus normal cells and exert cytotoxic effects preferentially on cancer cells, and that it could potentially be a valuable approach for therapy of pancreatic carcinomas."

The full text of the article is available here.